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\nThe suprachiasmatic nucleus (SCN) of the hypothalamus acts as the site of an endogenous rhythm of approximately 24 h in the mammalian circadian clock. In a study of clock gene expression, the novel gene product Period2 (Per2) has been localized to the SCN. The present studies examined the role of Per2 in the SCN circadian clock. A core circadian rhythm in PER2 expression was detected in several SCN tissue samples from hamsters of differing light:dark photoperiods. Expression levels were significantly reduced when hamsters were exposed to constant light. This reduction in PER2 expression was not due to a change in the amplitude of the rhythm, but rather to a change in the timing of the expression. In addition, examination of Per2 mRNA levels in the SCN using quantitative in situ hybridization showed a circadian oscillation of Per2 expression levels in the SCN that was inversely related to the expression levels of the established clock gene, Clock, and that was significantly reduced when constant light was imposed. Finally, daily injections of antisense oligonucleotides that targeted Per2 mRNA in the SCN abolished the circadian rhythm in PER2 expression, whereas sense oligonucleotides had no effect on Per2 expression. The data suggest a novel role for Per2 in the circadian clock of the SCN and a link between Per2 and the circadian expression of Per1 and Per2.Q:<\/p>\n
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