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Clinical observations have shown that approximately 50% of prostate cancer cases can spontaneously regress, and currently no information is available on the causes of the regression. We review several aspects of this problem with special emphasis on the molecular and biochemical changes leading to the regression of prostate cancer and on the possible implications for clinical practice. While it is known that the loss of androgen-dependent growth leads to regression, the molecular and biochemical changes that occur during regression remain unknown. Most of the molecular changes, which generally occur in the disease context of advanced androgen-independent tumors, could be associated with apoptosis, which occurs in the majority of cases at an early stage of the regression, often with a co-existing increase in the activity of specific antioxidant enzymes. Several of these changes, such as the increased activity of antioxidant enzymes (particularly catalase), could be potentially useful in selecting cancer patients that are most likely to respond to androgen deprivation or chemotherapy.TVB recently released their newest broadcast, the 2014 Chu’ a Chun�
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