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In Sweden, 1 million people are diagnosed with osteoporosis. The aim was to determine whether vitamin D intake/exercise status can predict future fractures in healthy women over 65 years of age. 2,668 women were recruited by random selection from the local population register in a city and a county, respectively. Vitamin D intake was self-reported by food frequency questionnaire and questionnaire about type and duration of exercise. VitD status was determined by measurement of serum 25-hydroxyvitamin D (25(OH)D). Bone mineral density (BMD) of the hip and spine was measured by dual-energy x-ray absorptiometry (DXA). The incidence of forearm, hip, and other fractures was determined from the regional patient register (Malmö). During a mean follow-up of 3 years, 229 women were admitted to hospital with a fragility fracture. In the unadjusted Cox regression analyses, no significant associations were found between vitamin D status and forearm fractures, but the hazard ratio (HR) for hip fracture was 1.6 (95% confidence interval, CI: 1.1-2.1) among the women with sufficient vitamin D and 2.4 (95% CI: 1.6-3.6) among those with vitamin D insufficiency. In the adjusted Cox regression analyses with additional adjustment for body mass index and bone status, the HR for hip fracture was increased to 2.7 (95% CI: 1.6-4.6) in the vitamin D insufficient group. Exercise did not confer any protective effect on hip fracture. The results do not indicate that vitamin D supplementation or exercise would prevent hip fractures in healthy elderly women. The results also support the contention that low 25(OH)D concentrations are a risk factor for hip fractures.Shown Here:Introduced in Senate (08/22/2012)
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The present study was undertaken to define the importance of HLA-A, B and BgG loci on the antigen-dependent K562 cell-inhibitory activity of natural killer (NK) cells purified from human peripheral blood. K562 and target cells were co-incubated with autologous effector cells in the presence or absence of monoclonal antibodies (mAbs) directed against HLA-A, B and BgG antigens. On the basis of cytotoxicity against K562 cells, NK cells were shown to recognize target cells via natural killer group specific (NKG) antigens. In this respect, we found that autologous NK cells were more efficient in killing K562 cells in comparison to allogeneic NK cells. In this context, K562 cells were more efficiently lysed by NK cells expressing HLA-A and B antigens with BgG restrictions than by NK cells lacking these determinants. Furthermore, mAbs directed against HLA-A2 and HLA-B8 revealed a selective inhibitory effect on NKG-dependent cytotoxicity of NK cells. Since these mAbs could specifically recognize HLA-A2,B8 and HLA-B7,8, specificity of HLA-B locus is important for NKG-dependent cytotoxicity of NK cells. In this respect, HLA-B7 and BgG antigens expressed on the surface of K562 cells appear to be important for NK-mediated lysis.Since the sub-theme of risk-management is an important part of the work our group has been engaged in during the past year, we have extensively studied the mechanisms that control the expression of the