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Julia Isabel Clara Simo Ebook 14
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Macrophage migration inhibitory factor (MIF), a cytokine associated with T-cell immunity, has been implicated in several autoimmune and infectious diseases. As this cytokine has been recently discovered in association with human hepatitis G virus (HGV), a non-retroviral single-stranded RNA virus, we explored its role in the pathogenesis of post-hepatitis G virus cirrhosis (HGV-CH). To evaluate the relationship between MIF and the progression of disease in HGV-CH, sera and liver tissue from a homogeneous cohort of 51 untreated HGV-CH patients were studied. Serum levels of MIF were markedly elevated in infected patients as compared with healthy subjects. Livers from patients with cirrhosis expressed high MIF levels in portal tracts. Portal tracts from cirrhotic livers with active hepatitis were immunohistochemically infiltrated with CD3+, CD4+, and CD8+ lymphocytes and MIF-producing cells. Moreover, TNF-alpha, IL-6, IL-1beta, and MIF immunoreactivities were more strongly expressed in portal areas than in centrilobular zones in the same biopsies. Finally, MIF serum levels correlated with the levels of TNF-alpha, IL-6, and IL-1beta, but not with those of other cytokines, in HGV-CH. These results suggest that the inflammatory process in liver tissue from HGV-CH patients is T-cell mediated and MIF might be involved in the pathogenesis of the disease, at least in part through the activation of proinflammatory cytokines.Q:
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Julia Isabel Clara Simo Ebook 14
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